Leucocyanidin reductase is protein in its nature.
无色花青素还原酶本质上是蛋白质。
Research prospects on nitrate reductase.
以及研究硝酸还原酶的展望。
Nitrate reductase is a short-lived enzyme.
硝酸还原酶是短寿酶。
Effects of a-naphthol on the glutathione and its reductase in Chlorella.
萘酚对小球藻谷胱甘肽及其还原酶的影响。
The NO level of lung homogenates was measured by Nitrate Reductase Test.
采用硝酸还原酶法测肺组织匀浆中NO含量。
The production of nitric oxide(NO) was determined by nitrate reductase method.
用硝酸还原酶法测一氧化氮（NO）的浓度。
Recent advances in azo-dye-degrading bacteria and azo reductase were reviewed.
综述了目前降解偶氮染料的细菌以及偶氮还原酶的研究进展。
Objective: To clone the thioredoxin reductase (TR) gene from human fatal brain.
目的：从人脑组织中克隆硫氧还蛋白还原酶基因。
Effect of different stress on roots activity and nitrate reductase activity in Zea mays L.
不同逆境对玉米幼苗根系活力及硝酸还原酶活性的影响。
Purpose: To study the QSAR of the ribonucleotide reductase inhibitors on the electron level.
目的：从电子水平上探讨具有抗肿瘤活性的核苷酸还原酶抑制剂的定量构效关系（QSAR）。
Biliverdin was biliverdin reductase reduced to bilirubin, and free iron and transferrin binding.
胆绿素随后被胆绿素还原酶还原成胆红素，而游离铁与转铁蛋白结合。
Aldose reductase (ar) plays an important role in sugar cataract and other complications of diabetes.
醛糖还原酶(AR)在糖性白内障和其它糖尿病并发症的发病中起着关键性的作用。
Objective To optimize the the NADPgenerating fluorimetric method for aldose reductase (AR) activity.
目的找出优化NADP生成荧光法测定红细胞醛糖还原酶(AR)活性的方案。
Aldose reductase is an enzyme that is normally present in the eye and in many other parts of the body.
醛式糖还原酶是通常是存在在眼睛和在许多其它身体部位的酵素。
This review mainly gives a brief account of the progress of research on aldose reductase inhibitors from microorganisms.
本文主要概述了微生物来源的醛糖还原酶抑制剂的研究进展。
This method has been applied to the design of inhibitors for E. coli dihydrofolate reductase and human phospholipase A2.
该方法已用于设计抑制剂大肠杆菌二氢叶酸还原酶和人类磷脂酶a2。
Results compared with the control group, the aldose reductase gene was down-regulated in the captopril intervention group.
结果与对照组相比，干预组醛糖还原酶基因表达下调。
The genetic polymorphisms due to mutations in the methylene tetrahydrofolate reductase gene may increase the risk for NTDs.
亚甲基四氢叶酸还原酶基因突变导致的遗传多态现象可增加神经管缺陷(ntd)发生的危险性。
Conclusion The K39 in HO has important effects on accepting and transporting electrons from NADPH-cytochrome P450 reductase.
结论HO的碱性氨基酸K39在接受并传递NADPH-细胞色素P450所提供的电子时具有重要作用。
Conclusions the increased expression of mitochondria thioredoxin reductase may protect the heart against oxidative stress injury.
结论线粒体型硫氧还蛋白还原酶基因和蛋白表达的增加有利于大鼠心肌抗氧化损伤。
Therefore, it is possible to use the activity of nitrate reductase as a parameter of diagnosing the nitrogen nutrition in sugarcane.
因此，把硝酸还原酶活力作为诊断蔗株氮素营养水平的指标是有可能的。
The activity of fumaric reductase in Cysticercus cellulosae tissue homogenate with albendazole and oxfendazole individually was detected.
研究测定了阿苯达唑和奥芬达唑对猪囊尾蚴组织匀浆延胡索酸还原酶活性的抑制作用。
The reducing potential of the NADH is utilized via the methemoglobin reductase pathway to maintain the iron in Hgb in its reduced form (Fe2 +).
高铁血红蛋白还原酶途径利用了NADH的还原电位维持血红蛋白中铁元素的还原态(Fe2 +)。
Objective To investigate the relationship between aldose reductase-like-1 (ARL-1) gene and drug resistance in human liver carcinoma cells.
目的探讨醛糖还原酶相似基因- 1 (ARL - 1基因)与人肝癌细胞耐药性的关系。
A novel screening method for dihydrofolate reductase inhibitors (DHFRI) using high performance capillary electrophoresis (HPCE) was developed.
建立了一种采用毛细管电泳法(CE)测定二氢叶酸还原酶(DHFR)反应动力学参数的新方法。
Results The expressions of the protein and gene of mitochondrial thioredoxin reductase in stress group were higher than those of control group.
结果应激组大鼠心肌中线粒体型硫氧还蛋白还原酶基因和蛋白表达均高于非应激组。
Objective To assess induction of quinone reductase (QR) and glutathions transferases (GSTs) by dimethyl fumarate (DMF) in major viscera of rats.
目的探讨延胡索酸二甲酯(DMF)对大鼠主要脏器醌还原酶和谷胱甘肽s转移酶的诱导作用和意义。
Overexpression of sterol C-24 (28) reductase gene ERG4 and sterol acyltransferase gene ARE2 in-creased the ergosterol biosynthesis in yeast cells.
发现参与麦角甾醇生物合成的甾醇c - 24(28)还原酶基因erg4和甾醇酰基转移酶基因ARE2高表达后，使细胞麦角甾醇含量提高。
Xylose reductase(XR) is a key enzyme in D-xylose metabolism, catalyzing the reduction of D-xylose to xylitol. The reported xylose reductase from C.
木糖还原酶（XR）是D-木糖代谢途径中的关键酶，催化还原D-木糖成木糖醇。
All cases were diagnosed as BH4 deficiency by analysis of urinary pterins profile, BH4 loading test, and determination of dihydropteridine reductase in RBC.
所有患者均经尿蝶呤谱分析、BH4口服负荷试验及红细胞二氢蝶呤还原酶测定，确诊为BH4缺乏症。
应用推荐